Arthur Kaser, MD PhD

University of Cambridge
Cambridge United Kingdom
Biographical Sketch:
Arthur Kaser is a mucosal immunologist and clinically trained gastroenterologist. Arthur has qualified from Leopold Franzens University Innsbruck with an intercalated thesis on type I interferons, and received post-doctoral training in Innsbruck and as a Max Kade and Schroedinger Fellow at Harvard Medical School’s Brigham & Women’s Hospital. Arthur’s laboratory at the University of Cambridge, where he holds the Chair in Gastroenterology, studies mechanisms of inflammation at the host – microbiota interface that are affected by genetic variation conferring risk for inflammatory bowel disease. His laboratory has a particular interest in the biology of the intestinal epithelium and the role of the unfolded protein response and autophagy within these cells. Arthur also serves as a clinical investigator and is involved in the design and delivery of early and late phase clinical trials in inflammatory bowel disease.

Any personal financial relationships? No

To the best of my ability, I will ensure that any speakers or content I suggest is independent of commercial bias.
I will recuse myself from planning activity content in which I have a conflict of interest.
1. I will insure balance, independence, objectivity, and scientific rigor in my role in the planning, development or presentation of this CME activity.
2. I will comply with the requirements to protect health information under the Health Insurance Portability & Accountability Act of 1996 (HIPAA).
3. I will disclose any discussion or reference to unapproved or unlabeled uses of therapeutic agents or products.
4. I have input my full name below as attestation of agreement with declaration statements. Marking the check box and indicating my name is in lieu of signature and is considered an "e-signature".

Signed on 06/30/2015 by Author Kaser
Papers:
Epithelial Stem Cells
The Mucosal Barrier
OR.9 B-Cells and Dendritic Cells React to Intestinal Epithelial Stress with a Coordinated and Protective IgA Response.